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The molecule that helps exercise protect the brain from Alzheimer’s - Arstechnica
Gives the phrase “muscle memory” a whole new meaning.
Sometimes data behaves so nicely, lining up just the way you want it to. In 2012, irisin was identified as a molecular messenger induced by exercise. In 2013, irisin was found to stimulate genes in the hippocampus, a region of the brain essential for making and storing memories. In 2017, epidemiological studies indicated that exercise could slow the development of Alzheimer’s disease (AD) and other kinds of dementia. And this week—you guessed it—new research demonstrated that exercise alleviates AD and slows memory loss by sending irisin to the brain.
Irisin is sent from muscles to various tissues throughout the body during exercise. It was initially found to promote fat metabolism, turning white fat cells into brown ones, which burn more energy. It was only later recognized that it also plays a role in the brain.
Gives the phrase “muscle memory” a whole new meaning.
Sometimes data behaves so nicely, lining up just the way you want it to. In 2012, irisin was identified as a molecular messenger induced by exercise. In 2013, irisin was found to stimulate genes in the hippocampus, a region of the brain essential for making and storing memories. In 2017, epidemiological studies indicated that exercise could slow the development of Alzheimer’s disease (AD) and other kinds of dementia. And this week—you guessed it—new research demonstrated that exercise alleviates AD and slows memory loss by sending irisin to the brain.
Irisin is sent from muscles to various tissues throughout the body during exercise. It was initially found to promote fat metabolism, turning white fat cells into brown ones, which burn more energy. It was only later recognized that it also plays a role in the brain.